Apparantly Creasy did some studies on this which is mentioned in the following aticle. However, I wouldn't expect any wines other than Pinot Noir to have significant resveratrol content...
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I'm looking for research showing resveratrol content of wines other than
> Pinot Noir and not grown in NY State. These studies (at bottom) have
> covered those bases.
>
> I'm hoping to find a high-resveratrol-content Shiraz, or something else
> that's chewier than (as per my perception) the thin and insubstantial Pinot > Noir.
>
> You on-topic responses would be appreciated.
> Thank you kindly.
>
>
>
> NY Wines Higest in Resveratrol
>
> The type of wine with by far the highest resveratrol levels was pinot noir,
> with 11 of the 17 New York wines registering above 10 µM. For pinot noir,
> the average levels were 13.6 µM for New York, 11 µM for all non-New York
> and 10.1 µM for California.
>
> Variations were striking in the cabernet sauvignon category. New York wine
> had an average of 8.3 µM in this category, with all non-New York cabernet
> sauvignons at 3.7 µM, and California's at 1.7 µM. New York cabernet
> sauvignon Francs averaged 8.6 µM, but non-New York wines were not analyzed
> for comparison in this category.
>
> New York merlots averaged 6.5 µM compared with all non-New York merlots at
> 4.7 µM and California merlots at 5.3 µM.
>
> New York wines also had the highest individual levels in each of these
> categories, with one merlot at 10.7 µM, a cabernet sauvignon at 19.2 µM, a
> pinot noir at 46.1 µM, a New York cabernet Franc at 16.9 µM and a lemberger
> at 15.3 µM.
>
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> --
>
> Nature. 425, 191-196, 2003.
>
> Next, we assessed whether resveratrol could activate SIRT1 in vivo.
> One known target of SIRT1 is lysine 382 of p53 (K382). Deacetylation of
> this residue by SIRT1 decreases the activity and half-life of p53, and
> increases cell survival under a variety of DNA damaging conditions8?10.
> Treatment of cells with a low concentration (0.5mM) of resveratrol
> increased cell survival after ionizing irradiation (Fig. 4b). To test
> whether this was due to modulation of SIRT1 activity, we
> generated an antibody that specifically recognizes the acetylated form of
> p53-K382 (Ac-K382) (Fig. 4c and data not shown). U2OS cells treated with
> 0.5mM resveratrol showed a marked decrease (about 75%) in the level of
> Ac-K382 (Fig. 4d; see also Supplementary Fig. 4a). At higher concentrations
> of resveratrol (.50mM) the effect was reversed (Fig. 4d and data not
> shown), which may explain the dichotomy in the literature regarding the
> effects of resveratrol on cell viability17,27,28.
> Treatment of HEK 293 cells with resveratrol led to a dose-dependent
> decrease in Ac-K382 levels that was diminished by overexpression of a
> dominantnegative SIRT1 allele (see Fig. 4e and Supplementary Fig. 4b).
> These data strongly indicate that resveratrol can promote cell survival by
> stimulating SIRT1 in vivo.
>
> ALSO SEE:
>
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